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Multilevel Plasticity and Altered Glycosylation Drive Aggressiveness in Hypoxic and Glucose-Deprived Bladder Cancer Cells
PubMed
Hypoxia dysregulates circadian rhythm and causes synapse elimination defects in astrocytes
Chronic Lymphocytic Leukemia in African Ancestry Population is Characterized by Increased Genomic Instability
TGF-β1-induced m6A modifications accelerate onset of nuclear cataract in high myopia by influencing the PCP pathway [meRIP-seq]
CD8α and CD70 mark human natural killer cell populations which differ in cytotoxicity
FBXW7 Mutations Reprograms Glucose Metabolism by Activating the ETV6-GLUT Axis in Endometrial Cancer
Enterochromaffin cells act as intestinal signal integration hubs for microbial metabolites
RNA-seq and IP-seq of Rbfox1(WT)/LASR and Rbfox1(F125A)/LASR
RNA sequencing of Flp-In 293 Rbfox2 KO#7 parental, Rbfox1(WT), and Rbfox1(F125A) expressing cells
Human adult high-plasticity stem cells generated by cell-cell communication
TFE3 fusion proteins drive oxidative metabolism, ferroptosis resistance and general RNA synthesis in translocation renal cell carcinoma
TFE3 fusion proteins drive oxidative metabolism, ferroptosis resistance and general RNA synthesis in translocation renal cell carcinoma. [RNA-seq]
TFE3 fusion proteins drive oxidative metabolism, ferroptosis resistance and general RNA synthesis in translocation renal cell carcinoma. [Cut and Tag/ChIP-seq]
Genetic effects on chromatin accessibility uncover mechanisms of liver gene regulation and quantitative traits
m6A modification governs the stability of both methylated and unmethylated mRNA isoforms across the transcriptome
Effects of m6A modification on turnover of newly synthesized mRNAs at the transcriptome level.
Transcriptome-based mapping of m6A sites in newly synthesized mRNA population in U2OS cells.
Effects of m6A modification and YTHDF2 knockdown on global mRNA turnover
Differential effects of m6A modification and YTHDF2 knockdown on global mRNA turnover
SRA Run Selector
Transcriptome-wide mapping of m6A sites with or without YTHDF2 (DF2) knock-down in U2OS cells.
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