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Items: 6

1.

Pancreatic cancer exosomes induce pre-metastatic niche formation in the liver

(Submitter supplied) Pancreatic cancers (PCs) are highly metastatic with poor prognosis, mainly due to delayed detection. We hypothesized that intercellular communication is critical for metastatic progression. Here, we show that PC-derived exosomes induce liver pre-metastatic niche formation in naïve mice and consequently increase liver metastatic burden. Uptake of PC-derived exosomes by Kupffer cells caused transforming growth factor β secretion and upregulation of fibronectin production by hepatic stellate cells. more...
Organism:
Homo sapiens
Type:
Expression profiling by high throughput sequencing
Platform:
GPL16791
4 Samples
Download data: TXT
Series
Accession:
GSE66876
ID:
200066876

PubMed Full text in PMC Similar studies Analyze with GEO2RSRA Run Selector

2.

Illumina HiSeq 2500 (Homo sapiens)

Organism:
Homo sapiens
11814 Series
336528 Samples
Download data
Platform
Accession:
GPL16791
ID:
100016791

3.

Human_Kupffer_Cells_BxPC3exo

Organism:
Homo sapiens
Source name:
Primary culture (Life technologies)
Platform:
GPL16791
Series:
GSE66876
Download data
Sample
Accession:
GSM1633691
ID:
301633691

SRA Run Selector

4.

Human_Kupffer_Cells_Control

Organism:
Homo sapiens
Source name:
Primary culture (Life technologies)
Platform:
GPL16791
Series:
GSE66876
Download data
Sample
Accession:
GSM1633690
ID:
301633690

SRA Run Selector

5.

PAN02EXO

Organism:
Homo sapiens
Source name:
Primary culture (Life technologies)
Platform:
GPL16791
Series:
GSE66876
Download data
Sample
Accession:
GSM1633689
ID:
301633689

SRA Run Selector

6.

CTCEXO

Organism:
Homo sapiens
Source name:
Primary culture (Life technologies)
Platform:
GPL16791
Series:
GSE66876
Download data
Sample
Accession:
GSM1633688
ID:
301633688

SRA Run Selector

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db=gds|term=GSE66876[Accession]|query=1|qty=2|blobid=MCID_66499440f6306928274fec31|ismultiple=true|min_list=5|max_list=20|def_tree=20|def_list=|def_view=|url=/Taxonomy/backend/subset.cgi?|trace_url=/stat?
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