GSM1071303[Accession] - GEO DataSets

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Items: 5

Select item 2000465031.

Selective suppression of endothelial cytokine production by progesterone receptor

(Submitter supplied) This SuperSeries is composed of the SubSeries listed below.

Organism:: Homo sapiens

Type:: Genome binding/occupancy profiling by high throughput sequencing; Expression profiling by high throughput sequencing

Platform:: GPL11154
14 Samples

Download data: BW, GTF, XLSX

Series

Accession:: GSE46503

ID:: 200046503

Select item 2000437892.

Physiological Vascular Permeability Requires Induction of Endothelial NR4A1 by Progesterone Receptor

(Submitter supplied) This SuperSeries is composed of the SubSeries listed below.

Organism:: Homo sapiens

Type:: Expression profiling by high throughput sequencing; Genome binding/occupancy profiling by high throughput sequencing

Platform:: GPL11154
10 Samples

Download data: BW, GTF, XLSX

Series

Accession:: GSE43789

ID:: 200043789

Select item 2000437863.

Physiological Vascular Permeability Requires Induction of Endothelial NR4A1 by Progesterone Receptor [ChIP-Seq]

(Submitter supplied) Vascular permeability is frequently associated with inflammation and it is triggered by chemokines and by a cohort of secreted permeability factors, such as VEGF. In contrast, here we showed that the physiological vascular permeability that precedes implantation is directly controlled by progesterone receptor (PR) and it is independent of VEGF. Both global and endothelial-specific deletion of PR block physiological vascular permeability in the uterus while misexpression of PR in the endothelium of other organs results in ectopic vascular leakage. more...