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Items: 3

1.

Mitochondrial stress induces chromatin reorganization to promote longevity and UPRmt

(Submitter supplied) Organisms respond to mitochondrial stress through the upregulation of an array of protective genes, often perpetuating an early response to metabolic dysfunction across a lifetime. We find that mitochondrial stress causes widespread changes in chromatin structure through histone H3K9 di-methylation marks traditionally associated with gene silencing. Mitochondrial stress response activation requires the di-methylation of histone H3K9 through the activity of the histone methyltransferase met-2 and the nuclear co-factor lin-65. more...
Organism:
Caenorhabditis elegans
Type:
Expression profiling by high throughput sequencing
Platform:
GPL18245
18 Samples
Download data: TXT
Series
Accession:
GSE78252
ID:
200078252

PubMed Full text in PMC Similar studies SRA Run Selector

2.

Illumina HiSeq 2500 (Caenorhabditis elegans)

Organism:
Caenorhabditis elegans
475 Series
8107 Samples
Download data
Platform
Accession:
GPL18245
ID:
100018245

3.

N2 cco-1 replicate 1

Organism:
Caenorhabditis elegans
Source name:
N2 cco-1_whole worm lysate
Platform:
GPL18245
Series:
GSE78252
Download data: TXT
Sample
Accession:
GSM2070428
ID:
302070428

SRA Run Selector

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db=gds|term=GSM2070428[Accession]|query=1|qty=2|blobid=MCID_69863602d44c2487b2e53dcc|ismultiple=true|min_list=5|max_list=20|def_tree=20|def_list=|def_view=|url=/Taxonomy/backend/subset.cgi?|trace_url=/stat?
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