PHF2 maintains neural progenitor genome stability by preserving pericentric heterochromatin integrity

ChIP–seq on periosteal cells derived from the pre-invasive group using antibodies to acetylated histone H3 Lys27 (H3K27ac), H3K4me3 and HIF-1α

FOXC1 in triple negative breast cancer [ChIP-seq]

FOXC1 in triple negative breast cancer

Enhanced crosslinking and immunoprecipitation analysis of human fetal β-cells

A PRE loop at the dac locus acts as a topological chromatin structure that restricts and specifies enhancer promoter communication

A CARM1 inhibitor potently suppresses breast cancer both in vitro and in vivo

ARID1A regulates DSB repair through chromatin organization and its deficiency triggers DNA damage-mediated immune response [ATAC-seq]

ARID1A regulates DSB repair through chromatin organization and its deficiency triggers DNA damage-mediated immune response

USP44 overexpression drives a MYC-like gene expression program in neuroblastoma through epigenetic reprogramming

Myb is regulated by -81 enhancer eRNA transcript Myrlin through recruitment of MLL1 complex

A Constitutive Heterochromatic Region Shapes Genome Organization and Impacts Gene Expression in Neurospora crassa [ChIP-Seq]

A Constitutive Heterochromatic Region Shapes Genome Organization and Impacts Gene Expression in Neurospora crassa

Novel O-GlcNAcylation on Thr71 of histone H4 preferentially localizes to active gene regions

Histone serotonylation regulates ependymoma tumorigenesis through neurodevelopmental pathways [ChIP-Seq]

A two-step regulatory mechanism dynamically controls histone H3 acetylation by SAGA complex at growth-related promoters

Contribution of CENP-F to FOXM1-mediated discordant centromere and kinetochore transcriptional regulation [ATAC-seq]

Contribution of CENP-F to FOXM1-mediated discordant centromere and kinetochore transcriptional regulation. [CHIP-Seq]

Contribution of CENP-F to FOXM1-mediated discordant centromere and kinetochore transcriptional regulation

Elevating PLK1 Overcomes BETi-Resistance in Prostate Cancer via Triggering BRD4 Phosphorylation-dependent Degradation in Mitosis [ChIP-seq]