GPL29397[Accession] - GEO DataSets - NCBI

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Select item 2002461021.

Calprotectin blockade inhibits long-term vascular pathology following peritoneal dialysis-associated bacterial infection.

(Submitter supplied) This SuperSeries is composed of the SubSeries listed below.

Organism:: Mus musculus

Type:: Expression profiling by RT-PCR

Platform:: GPL29397
7 Samples

Series

Accession:: GSE246102

ID:: 200246102

PubMed Full text in PMC Similar studies

Select item 2002461012.

Calprotectin blockade inhibits long-term vascular pathology following peritoneal dialysis-associated bacterial infection. [2]

(Submitter supplied) Bacterial infections and the concurrent inflammation have been associated with increased long-term cardiovascular (CV) risk. In patients receiving peritoneal dialysis (PD), bacterial peritonitis is a common occurrence, and each episode further increases late CV mortality risk. However, the underlying mechanism(s) remains to be elucidated before safe and efficient anti-inflammatory interventions can be developed. more...

Organism:: Mus musculus

Type:: Expression profiling by RT-PCR

Platform:: GPL29397
4 Samples

Download data: XLS

Series

Accession:: GSE246101

ID:: 200246101

PubMed Full text in PMC Similar studies

Select item 2002461003.

Calprotectin blockade inhibits long-term vascular pathology following peritoneal dialysis-associated bacterial infection. [1]

(Submitter supplied) Bacterial infections and the concurrent inflammation have been associated with increased long-term cardiovascular (CV) risk. In patients receiving peritoneal dialysis (PD), bacterial peritonitis is a common occurrence, and each episode further increases late CV mortality risk. However, the underlying mechanism(s) remains to be elucidated before safe and efficient anti-inflammatory interventions can be developed. more...

Organism:: Mus musculus

Type:: Expression profiling by RT-PCR

Platform:: GPL29397
3 Samples

Download data: XLS

Series

Accession:: GSE246100

ID:: 200246100

PubMed Full text in PMC Similar studies

Select item 2002361954.

Therapeutic targeting of Chronic Kidney Disease-associated DAMPs differentially contributing to vascular pathology.

(Submitter supplied) This SuperSeries is composed of the SubSeries listed below.

Organism:: Mus musculus

Type:: Expression profiling by RT-PCR

Platforms:: GPL23971 GPL29397
10 Samples

Series

Accession:: GSE236195

ID:: 200236195

PubMed Full text in PMC Similar studies

Select item 2002361945.

Therapeutic targeting of Chronic Kidney Disease-associated DAMPs differentially contributing to vascular pathology. [atherosclerosis AAN+Paquinimod aortas]

(Submitter supplied) Chronic Kidney Disease (CKD) is associated with markedly increased cardiovascular (CV) morbidity and mortality. Chronic inflammation, a hallmark of both CKD and CV diseases (CVD), is believed to drive this association. Pro-inflammatory TLR agonists, Damage-Associated Molecular Patterns (DAMPs), have been found elevated in CKD patients’ plasma and suggested to promote CVD, however, confirmation of their involvement, the underlying mechanism(s), the extent to which individual DAMPs contribute to vascular pathology in CKD and the evaluation of potential therapeutic strategies, have remained largely undescribed. more...

Organism:: Mus musculus

Type:: Expression profiling by RT-PCR

Platform:: GPL29397
3 Samples

Download data: XLSX

Series

Accession:: GSE236194

ID:: 200236194

PubMed Full text in PMC Similar studies

Select item 2002361926.

Therapeutic targeting of Chronic Kidney Disease-associated DAMPs differentially contributing to vascular pathology. [atherosclerosis AAN+sTLR2 aortas]

(Submitter supplied) Chronic Kidney Disease (CKD) is associated with markedly increased cardiovascular (CV) morbidity and mortality. Chronic inflammation, a hallmark of both CKD and CV diseases (CVD), is believed to drive this association. Pro-inflammatory TLR agonists, Damage-Associated Molecular Patterns (DAMPs), have been found elevated in CKD patients’ plasma and suggested to promote CVD, however, confirmation of their involvement, the underlying mechanism(s), the extent to which individual DAMPs contribute to vascular pathology in CKD and the evaluation of potential therapeutic strategies, have remained largely undescribed. more...

Organism:: Mus musculus

Type:: Expression profiling by RT-PCR

Platform:: GPL29397
3 Samples

Download data: XLSX

Series

Accession:: GSE236192

ID:: 200236192

PubMed Full text in PMC Similar studies

Select item 2001612167.

Effects of TREM-1 pharmacological blockade within aortas of apoE KO mice

(Submitter supplied) mouse aorta

Organism:: Mus musculus

Type:: Expression profiling by RT-PCR

Platform:: GPL29397
4 Samples

Download data: TXT

Series

Accession:: GSE161216

ID:: 200161216

Analyze with GEO2R

Select item 1000293978.

RT² Profiler™ PCR Array Mouse Atherosclerosis

(Submitter supplied) RT² Profiler™ PCR Array Mouse Atherosclerosis Protocol: see manufacturer's website

Organism:: Mus musculus

7 Series
17 Samples

Platform

Accession:: GPL29397

ID:: 100029397

Select item 3078554679.

PDF-exposed mice + S.epidermidis peritonitis

Organism:: Mus musculus

Source name:: aortas

Platform:: GPL29397
Series:: GSE246101 GSE246102

Sample

Accession:: GSM7855467

ID:: 307855467

Select item 30785546610.

Non infected PDF-exposed mice (i.p. catheter fitter, daily PDF administration)

Organism:: Mus musculus

Source name:: aortas

Platform:: GPL29397
Series:: GSE246101 GSE246102

Sample

Accession:: GSM7855466

ID:: 307855466

Select item 30785546511.

Control + S.epidermidis peritonitis

Organism:: Mus musculus

Source name:: aortas

Platform:: GPL29397
Series:: GSE246101 GSE246102

Sample

Accession:: GSM7855465

ID:: 307855465

Select item 30785546412.

Non-infected Control mice

Organism:: Mus musculus

Source name:: aortas

Platform:: GPL29397
Series:: GSE246101 GSE246102

Sample

Accession:: GSM7855464

ID:: 307855464

Select item 30785546313.

S.epidermidis-injected mice with Paquinimod treatment

Organism:: Mus musculus

Source name:: aortas

Platform:: GPL29397
Series:: GSE246100 GSE246102

Sample

Accession:: GSM7855463

ID:: 307855463

Select item 30785546214.

S.epidermidis-injected mice

Organism:: Mus musculus

Source name:: aortas

Platform:: GPL29397
Series:: GSE246100 GSE246102

Sample

Accession:: GSM7855462

ID:: 307855462

Select item 30785546115.

Control mice sample

Organism:: Mus musculus

Source name:: aortas

Platform:: GPL29397
Series:: GSE246100 GSE246102

Sample

Accession:: GSM7855461

ID:: 307855461

Select item 30751958316.

AAN mice with Paquinimod treatment

Organism:: Mus musculus

Source name:: aortas

Platform:: GPL29397
Series:: GSE236194 GSE236195

Sample

Accession:: GSM7519583

ID:: 307519583

Select item 30751958217.

Aristolochic Acid -induced Nephropathic (ANN) mice.

Organism:: Mus musculus

Source name:: aortas

Platform:: GPL29397
Series:: GSE236194 GSE236195

Sample

Accession:: GSM7519582

ID:: 307519582

Select item 30751958118.

Organism:: Mus musculus

Source name:: aortas

Platform:: GPL29397
Series:: GSE236194 GSE236195

Sample

Accession:: GSM7519581

ID:: 307519581

Select item 30751957619.

AAN mice with soluble TLR2 treatment

Organism:: Mus musculus

Source name:: aortas

Platform:: GPL29397
Series:: GSE236192 GSE236195

Sample

Accession:: GSM7519576

ID:: 307519576

Select item 30751957520.

Aristolochic Acid -induced Nephropathic (ANN) mice

Organism:: Mus musculus

Source name:: aortas

Platform:: GPL29397
Series:: GSE236192 GSE236195

Sample

Accession:: GSM7519575

ID:: 307519575

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