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Modeling pancreatic beta cell senescence by induction of DNA double-strand breaks
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Characterization of EndoC-BH5 a novel human pancreatic beta cell line
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Characterization of EndoC-BH5 a novel human pancreatic beta cell line. Comparative expression profile after stimulation stimulation with Interferon gamma and interleukin 1b.
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Characterization of EndoC-BH5 a novel human pancreatic beta cell line. Comparative expression profile with EndoC-BH1 and effect of stimulation with Interferon gamma and interleukin 1b.
Targeted elimination of senescent beta cells prevents Type 1 Diabetes
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Unique human beta-cell senescence-associated secretory phenotype (SASP) reveal conserved signaling pathways and heterogeneous factors
SASP signature of beta-cells includes pro-inflammatory and stress response proteins
Senescent beta-cells exhibit a unique secretory phenotype that promotes inflammation and remodeling of the extracellular environment
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Decreased IGF1R Attenuates Senescence and Improves Function in Pancreatic beta-Cells
Epigenetic modulation of β-cells by interferon-α via PNPT11-miR-26a-TET2 triggers autoimmune diabetes
Epigenetic modulation of β-cells by interferon-α via PNPT11-miR-26a-TET2 triggers autoimmune diabetes [RNA-seq]
Epigenetic modulation of β-cells by interferon-α via PNPT11-miR-26a-TET2 triggers autoimmune diabetes [methylation array]
Characterisation of the functional and transcriptomic effects of pro-inflammatory cytokines on human EndoC-βH5 beta cells
Inhibition of 12/15-Lipoxygenase Protects Against β Cell Oxidative Stress and Glycemic Deterioration in Mouse Models of Type 1 Diabetes
PAHSAs reduce cellular senescence and protect pancreatic beta cells from metabolic stress through regulation of Mdm2/p53
Early senescence-induced immunosuppressive β-cell secretome prevents type 1 diabetes
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RNA sequencing of control and PTPN2 knocked down transcriptomes in EndoC- H1 cells with or without the treatment of pro-inflammatory cytokines
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Alpha, beta and delta cells reponse to IFNγ
Gene expression of PD-L1 high and low pancreatic beta cells, after 20h of incubation wiht 50pg.mL of IFNγ.
Loss of tetraspanin-7 expression reduces pancreatic beta-cell exocytosis Ca2+ sensitivity but has limited effect on systemic metabolism
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