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Series GSE83788 Query DataSets for GSE83788
Status Public on Nov 15, 2017
Title SMAD2/3 are redirected to novel sites in MCF10A MII after prolonged TGFβ stimulation
Organism Homo sapiens
Experiment type Expression profiling by high throughput sequencing
Genome binding/occupancy profiling by high throughput sequencing
Summary This SuperSeries is composed of the SubSeries listed below.
 
Overall design Refer to individual Series
 
Citation(s) 29186616
Submission date Jun 27, 2016
Last update date Jul 25, 2021
Contact name Masato Morikawa
E-mail(s) morikawa-tky@umin.ac.jp
Phone +81-3-3964-1211
Organization name Teikyo University
Department Advanced Comprehensive Research Organization
Street address 2-21-1 Kaga, Itabashi-ku
City Tokyo
ZIP/Postal code 173-0003
Country Japan
 
Platforms (4)
GPL10999 Illumina Genome Analyzer IIx (Homo sapiens)
GPL11154 Illumina HiSeq 2000 (Homo sapiens)
GPL16791 Illumina HiSeq 2500 (Homo sapiens)
Samples (18)
GSM2218826 MCF10A MII, control_RNA
GSM2218827 MCF10A MII, TGFβ 1.5h_RNA
GSM2218828 MCF10A MII, TGFβ 16h_RNA
This SuperSeries is composed of the following SubSeries:
GSE83786 JUNB is a critical AP1 component for SMAD2/3 binding after TGFβ stimulation [RNA-seq]
GSE83787 SMAD2/3 binding pattern in a breast epithelial cell line, MCF10A MII [ChIP-seq]
GSE104352 SMAD2/3 binding pattern in triple-negative breast cancer cell lines, Hs578T and BT549 [ChIP-seq]
Relations
BioProject PRJNA326978

Download family Format
SOFT formatted family file(s) SOFTHelp
MINiML formatted family file(s) MINiMLHelp
Series Matrix File(s) TXTHelp

Supplementary file Size Download File type/resource
GSE83788_RAW.tar 1.2 Gb (http)(custom) TAR (of BED, BW)
SRA Run SelectorHelp

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