|
Status |
Public on Mar 07, 2017 |
Title |
ICF-specific DNMT3B dysfunction interferes with intragenic regulation of mRNA transcription and alternative splicing |
Organism |
Homo sapiens |
Experiment type |
Expression profiling by high throughput sequencing Other Genome binding/occupancy profiling by high throughput sequencing Methylation profiling by high throughput sequencing
|
Summary |
This SuperSeries is composed of the SubSeries listed below.
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|
|
Overall design |
Refer to individual Series
|
|
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Citation(s) |
28334849 |
Submission date |
Mar 06, 2017 |
Last update date |
May 15, 2019 |
Contact name |
Miriam Gagliardi |
E-mail(s) |
miriam_gagliardi@psych.mpg.de
|
Organization name |
Max Planck Institute of Psychiatry
|
Street address |
Kraepelinstrasse 2-10
|
City |
Munich |
State/province |
Bayern |
ZIP/Postal code |
80804 |
Country |
Germany |
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|
Platforms (2) |
GPL11154 |
Illumina HiSeq 2000 (Homo sapiens) |
GPL13393 |
AB SOLiD 4 System (Homo sapiens) |
|
Samples (40)
|
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This SuperSeries is composed of the following SubSeries:
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GSE95743 |
ICF-specific DNMT3B dysfunction interferes with intragenic regulation of mRNA transcription and alternative splicing (RNA-seq) |
GSE95744 |
ICF-specific DNMT3B dysfunction interferes with intragenic regulation of mRNA transcription and alternative splicing (RIP-seq) |
GSE95745 |
ICF-specific DNMT3B dysfunction interferes with intragenic regulation of mRNA transcription and alternative splicing (RRBS-Seq) |
GSE95746 |
ICF-specific DNMT3B dysfunction interferes with intragenic regulation of mRNA transcription and alternative splicing (ChIP-Seq) |
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Relations |
BioProject |
PRJNA378257 |