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Changes in chromatin state reveal a central role for the transcription factor ARNT2, in the control of glioblastoma stem cell tumorigenicity
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Epigenomic profiling of stemness, differentiation and primary tissues in human glioblastoma
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Reconstructing and reprogramming the tumor propagating potential of glioblastoma stem-like cells: RNA-seq
Reconstructing and reprogramming the tumor propagating potential of glioblastoma stem-like cells: ChIP-seq
Epigenetic regulation of MYC modulates glioblastoma tumorigenicity
Expression data from U87MG subclones
PubMed Full text in PMC Similar studies Analyze with GEO2R
Identifying ASCL1-mediated chromatin changes in primary GBM stem cell cultures [ATAC-seq]
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Temporal gene expression of human-fetal and glioblastoma stem cell cultures under directed differentiation conditions
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Identifying ASCL1 target genes in primary GBM stem cell cultures [ChIP-seq]
Identifying ASCL1 target genes in primary GBM stem cell cultures [RNA-seq]
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ASCL1 mediates neuronal differentiation of primary GBM stem cell cultures upon Notch signalling blockade [RNA-seq]
Bivalent chromatin domains in glioblastoma multiforme reveal an epigenetic signature of early neural development mediated by SHH and Wnt signaling
Next Generation Sequencing Facilitates Quantitative Analysis of Wild Type and TWIST1 knock out U87 xenograft mice transcriptomes
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RNAseq transcriptomic profile of glioblastoma stem-like cells derived from U87MG cell line treated with a selective A3 adenosine receptor antagonist (MRS1220) under hypoxia
ChIP-chip and MeDIP-chip from glioblastoma BTSCs (brain tumor stem cells) with H3K4me3, H3K27me3, H3K9me3, methylated DNA
Expression data of J3T-1 and J3T-2 glioma
Epigenetic determinants of self-renewal in glioblastoma [ATAC-seq]
MLL5 Orchestrates a Cancer Self-Renewal State by Repressing the Histone Variant H3.3 and Globally Reorganizing Chromatin [expression]
MLL5 orchestrates a cancer self-renewal state by repressing the histone variant H3.3 and globally reorganizing chromatin [methylation]
EGFR Mutation Promotes Glioblastoma Through Epigenome and Transcription Factor Network Remodeling
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