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Expression data of iPS microglia treated with TREM2 agonist antibody
PubMed Full text in PMC Similar studies Analyze with GEO2RSRA Run Selector
Transcriptomic and functional deficits in human TREM2-/- microglia impair response to Alzheimer's pathology in vivo
PubMed Full text in PMC Similar studies
Transcriptomic and functional deficits in human TREM2-/- microglia impair response to Alzheimer's pathology in vivo [bulk RNA-seq]
PubMed Full text in PMC Similar studies SRA Run Selector
Transcriptomic and functional deficits in human TREM2-/- microglia impair response to Alzheimer's pathology in vivo [scRNA-seq]
Transcriptomic and functional deficits in human TREM2-/- microglia impair response to Alzheimer’s pathology in vivo [RNA-seq]
Prior activation state shapes the microglia response to anti-human TREM2 in a mouse model of Alzheimer's disease
CD22 blockage restores age-related impairments of microglia surveillance capacity
Generation and characterization of human iPSC-derived microglia-like cells for the use in drug discovery
Human and mouse single-nucleus transcriptomics reveal TREM2-dependent and -independent cellular responses in Alzheimer’s disease
Human and mouse single-nucleus transcriptomics reveal TREM2-dependent and -independent cellular responses in Alzheimer’s disease [7 months]
Human and mouse single-nucleus transcriptomics reveal TREM2-dependent and -independent cellular responses in Alzheimer’s disease [15 months]
TREM2 Acts Downstream of CD33 in Modulating Microglial Pathology in Alzheimer's Disease
Off target expression data from iPSC derived microglia treated with APOE/TREM2 ASOs for 24h/48h. The iPSC cells are from a wild type donor (BIONi10C).
PubMed Full text in PMC Similar studies Analyze with GEO2R
Alzheimer’s disease neuroinflammatory risk genes can be targeted with RNase-H active antisense oligonucleotides in human microglia
The TREM2-APOE pathway drives the transcriptional phenotype of dysfunctional microglia in neurodegenerative diseases VI
The TREM2-APOE pathway drives the transcriptional phenotype of dysfunctional microglia in neurodegenerative diseases V
The TREM2-APOE pathway drives the transcriptional phenotype of dysfunctional microglia in neurodegenerative diseases IV
The TREM2-APOE pathway drives the transcriptional phenotype of dysfunctional microglia in neurodegenerative diseases
The TREM2-APOE pathway drives the transcriptional phenotype of dysfunctional microglia in neurodegenerative diseases III
The TREM2-APOE pathway drives the transcriptional phenotype of dysfunctional microglia in neurodegenerative diseases II
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